tag:blogger.com,1999:blog-19898266.post5299812372064991104..comments2023-08-14T07:57:55.501-05:00Comments on Wildlife Disease News Digest: Wildlife Data Integration Network [WDIN]http://www.blogger.com/profile/14944604969357532263noreply@blogger.comBlogger1125tag:blogger.com,1999:blog-19898266.post-36938648101241513212007-07-30T16:44:00.000-05:002007-07-30T16:44:00.000-05:00Greetings,IN reply to ;Cuts would reduce CWD budge...Greetings,<BR/><BR/>IN reply to ;<BR/><BR/>Cuts would reduce CWD budget by 60 percent<BR/><BR/>Published Monday, July 23, 2007 11:59:09 AM Central Time<BR/>By Lee Fahrney Times Outdoors Writer <BR/><BR/><BR/>http://www.themonroetimes.com/m0723cwd.htm<BR/><BR/><BR/><BR/>THIS disturbs me greatly, and should disturb every hunter in Wisconsin, and neighboring states. With CWD spreading like it is, we should not be<BR/>cutting any funding for CWD by 60 %. It makes no sense, with CWD amplifying and spreading still. <BR/><BR/><BR/>193 CONFIRMED POSITIVE CWD samples from 19,953 samples collected last time in Wisconsin, and they want to reduce funding by 60% for CWD management activities ???<BR/><BR/>this is not logical ???<BR/><BR/>do they now think that CWD is under control in Wisconsin ???<BR/><BR/>do they think that CWD is now NOT a potential threat to animal and or human health, and or the environment ???<BR/><BR/>what is the logistics behind this decision ???<BR/><BR/><BR/>2006-2007 Harvest and CWD Testing<BR/>CWD Public Dialogue to Begin<BR/><BR/>Preliminary numbers show that during the<BR/>2006-07 deer seasons, 56,593 deer were<BR/>harvested from the CWD zones. This is a<BR/>decline of more than 15 percent from the 2005-<BR/>06 season. Antlerless harvest accounts for<BR/>this drop, having decreased 31 percent from<BR/>last year. Buck harvest, however, increased<BR/>by five percent.<BR/>Of the 19,953 samples collected last<BR/>season, 193 tested CWD-positive. Thirty-five<BR/>of these deer were harvested in the herd<BR/>reduction zone. The rest were harvested in<BR/>the disease eradication zones.<BR/>This brings the total number of free ranging<BR/>CWD-positive deer to 844 for all years. Fiftyfour<BR/>of these deer were harvested in the herd<BR/>reduction zone.<BR/>As part of the effort to confirm that CWD<BR/>has not spread out of the CWD zones of southern<BR/>Wisconsin, 9,308 samples were also<BR/>collected in west-central Wisconsin and areas<BR/>of south-central and south-east Wisconsin not<BR/>in a CWD zone. None of these samples tested<BR/>positive for the disease.<BR/><BR/><BR/>http://dnr.wi.gov/org/land/wildlife/whealth/issues/cwd/doc/07_05_news.pdf<BR/><BR/><BR/>CWD Update 87<BR/><BR/>July 6, 2007<BR/><BR/>State and Provincial Updates<BR/><BR/>Illinois:<BR/><BR/>Paul Shelton, Illinois Department of Natural Resources provides the<BR/>following: Illinois<BR/><BR/><BR/>Department of Natural Resources staff collected 6,733 usable CWD surveillance samples during the period beginning July 1, 2006. <BR/>A few additional samples from suspect deer remain untested at this time. Testing yielded 41 CWD-positive deer and 6,692 ‘not detected’.<BR/>Forty-one positives were detected in four counties in northern Illinois: Winnebago (18), Boone (13), DeKalb (6), and McHenry (4). No <BR/>new counties were identified as having CWD, and no disease was detected in Ogle County, in which CWD was first identified during the <BR/>2005-2006 sampling season. However, CWD was identified in southeastern DeKalb County, more than 20 miles southeast of <BR/>previously-identified locations. Samples were collected from a variety of sources, including deer check stations in high-risk counties (3,097), <BR/>sample drop-off locations for archery hunters (175), cooperating meat processors (1,778), suspect deer (25), roadkills in CWD counties<BR/>(16), and culling efforts (1,642). Cooperating meat lockers were added as a sampling source this year to provide a statewide sampling base <BR/>outside the identified CWD area. Hunter-harvested deer accounted for 17 (41%) of the positives identified, with the remainder coming from <BR/>suspect deer (4; 10%); roadkills (1; 2%); and sharpshooting (19; 46%). Illinois DNR CWD information is available at: <BR/><BR/>http://dnr.state.il.us/cwd.<BR/><BR/><BR/>http://wildlifedisease.nbii.gov/documents/CWD%20Updates/Update%2087.pdf<BR/><BR/><BR/><BR/>Title: Susceptibility of cattle to first-passage intracerebral inoculation with chronic wasting disease agent from white-tailed deer <BR/><BR/><BR/>http://www.ars.usda.gov/research/publications/Publications.htm?seq_no_115=194089<BR/><BR/><BR/><BR/>Title: Transmission of chronic wasting disease of mule deer to Suffolk sheep following intracerebral inoculation <BR/><BR/>Submitted to: Journal of Veterinary Diagnostic Investigation <BR/>Publication Type: Peer Reviewed Journal <BR/>Publication Acceptance Date: June 20, 2006 <BR/>Publication Date: November 1, 2006 <BR/>Citation: Hamir, A.N., Kunkle, R.A., Cutlip, R.C., Miller, J.M., Williams, E.S., Richt, J.A. 2006. Transmission of chronic wasting disease of mule deer to Suffolk sheep following intracerebral inoculation. Journal of Veterinary Diagnostic Investigation. 18(6):558-565. <BR/><BR/>Interpretive Summary: Chronic wasting disease (CWD) has been identified in captive and free ranging deer and elk since 1967. To determine the transmissibility of CWD to sheep and to provide information about the disease and tests for detection of CWD in sheep, 8 lambs were inoculated with brain suspension from mule deer naturally affected with CWD. Two other lambs were kept as controls. Only 1 sheep developed clinical disease at 35 months after inoculation. The study was terminated at 72 months after the inoculation. At that time one other sheep was found to be positive for the disease. It is proposed that the host's genetic makeup may play a role in transmission of the disease to domestic sheep. Impact. This is the first study which shows that it is possible to transmit CWD to a small number of sheep. <BR/>Technical Abstract<BR/><BR/><BR/>snip...end<BR/><BR/>http://www.ars.usda.gov/research/publications/publications.htm?SEQ_NO_115=191571<BR/><BR/><BR/><BR/>0022-538X/05/$08.00+0 doi:10.1128/JVI.79.21.13794-13796.2005<BR/>Copyright © 2005, American Society for Microbiology. All Rights Reserved. <BR/><BR/>Interspecies Transmission of Chronic Wasting Disease Prions to Squirrel Monkeys (Saimiri sciureus) <BR/>Richard F. Marsh,1, Anthony E. Kincaid,2 Richard A. Bessen,3 and Jason C. Bartz4* <BR/>Department of Animal Health and Biomedical Sciences, University of Wisconsin, Madison 53706,1 Department of Physical Therapy,2 Department of Medical Microbiology and Immunology, Creighton University, Omaha, Nebraska 68178,4 Department of Veterinary Molecular Biology, Montana State University, Bozeman, Montana 597183 <BR/><BR/>Received 3 May 2005/ Accepted 10 August 2005 <BR/><BR/>Chronic wasting disease (CWD) is an emerging prion disease of deer and elk. The risk of CWD transmission to humans following exposure to CWD-infected tissues is unknown. To assess the susceptibility of nonhuman primates to CWD, two squirrel monkeys were inoculated with brain tissue from a CWD-infected mule deer. The CWD-inoculated squirrel monkeys developed a progressive neurodegenerative disease and were euthanized at 31 and 34 months postinfection. Brain tissue from the CWD-infected squirrel monkeys contained the abnormal isoform of the prion protein, PrP-res, and displayed spongiform degeneration. This is the first reported transmission of CWD to primates. <BR/><BR/><BR/>--------------------------------------------------------------------------------<BR/><BR/>* Corresponding author. Mailing address: Department of Medical Microbiology and Immunology, Creighton University, 2500 California Plaza, Omaha, NE 68178. Phone: (402) 280-1811. Fax: (402) 280-1875. E-mail: jbartz@creighton.edu . <BR/><BR/>Deceased. <BR/><BR/><BR/>--------------------------------------------------------------------------------<BR/><BR/>Journal of Virology, November 2005, p. 13794-13796, Vol. 79, No. 21<BR/>0022-538X/05/$08.00+0 doi:10.1128/JVI.79.21.13794-13796.2005<BR/>Copyright © 2005, American Society for Microbiology. All Rights Reserved. <BR/><BR/><BR/>http://jvi.asm.org/cgi/content/abstract/79/21/13794?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=cwd&searchid=1129736446553_4280&stored_search=&FIRSTINDEX=0&volume=79&issue=21&journalcode=jvi<BR/><BR/><BR/><BR/>Chronic Wasting Disease and Potential Transmission to Humans<BR/>Ermias D. Belay,* Ryan A. Maddox,* Elizabeth S. Williams,† Michael W. Miller,‡ Pierluigi Gambetti,§ and Lawrence B. Schonberger*<BR/>*Centers for Disease Control and Prevention, Atlanta, Georgia, USA; †University of Wyoming, Laramie, Wyoming, USA; ‡Colorado Division of Wildlife, Fort Collins, Colorado, USA; and §Case Western Reserve University, Cleveland, Ohio, USA<BR/><BR/>Suggested citation for this article: Belay ED, Maddox RA, Williams ES, Miller MW, Gambetti P, Schonberger LB. Chronic wasting disease and potential transmission to humans. Emerg Infect Dis [serial on the Internet]. 2004 Jun [date cited]. Available from: http://www.cdc.gov/ncidod/EID/vol10no6/03-1082.htm<BR/><BR/><BR/>--------------------------------------------------------------------------------<BR/><BR/>Chronic wasting disease (CWD) of deer and elk is endemic in a tri-corner area of Colorado, Wyoming, and Nebraska, and new foci of CWD have been detected in other parts of the United States. Although detection in some areas may be related to increased surveillance, introduction of CWD due to translocation or natural migration of animals may account for some new foci of infection. Increasing spread of CWD has raised concerns about the potential for increasing human exposure to the CWD agent. The foodborne transmission of bovine spongiform encephalopathy to humans indicates that the species barrier may not completely protect humans from animal prion diseases. Conversion of human prion protein by CWD-associated prions has been demonstrated in an in vitro cell-free experiment, but limited investigations have not identified strong evidence for CWD transmission to humans. More epidemiologic and laboratory studies are needed to monitor the possibility of such transmissions.<BR/><BR/><BR/>http://www.cdc.gov/ncidod/EID/vol10no6/03-1082.htm<BR/><BR/><BR/><BR/>May 2007<BR/><BR/>NIAID Research on Prion Diseases<BR/>OVERVIEW<BR/><BR/>snip...<BR/><BR/>More research is necessary to determine whether CWD poses any risk to<BR/>humans, particularly because it is spreading over a wider geographical area<BR/>in the United States. There have been several reported cases of CJD in<BR/>individuals who have consumed venison, most much younger than the typical<BR/>age associated with CJD. In each of these instances, careful investigations<BR/>by CDC have shown no causal link between CJD and CWD in deer and elk<BR/>populations. Continued surveillance is important, however, to assess any<BR/>possible risk of CWD transmission to humans.<BR/><BR/><BR/><BR/>http://www.niaid.nih.gov/factsheets/priondis.htm<BR/><BR/><BR/><BR/>> In each of these instances, careful investigations by CDC have shown no _causal_ link between CJD and CWD in deer and elk populations. <BR/><BR/><BR/>WRONG !<BR/><BR/>BELAY ET AL STATES ;<BR/><BR/>''Our conclusion stating that we found no _strong_ evidence of CWD transmission to humans'' <BR/><BR/><BR/>THERE'S a big difference between ''casual'' and ''strong''. ...TSS<BR/><BR/><BR/>Colorado Surveillance Program for Chronic Wasting Disease<BR/>Transmission to Humans (TWO SUSPECT CASES)<BR/><BR/><BR/><BR/>From: TSS (216-119-163-189.ipset45.wt.net)<BR/>Subject: CWD aka MAD DEER/ELK TO HUMANS ???<BR/>Date: September 30, 2002 at 7:06 am PST<BR/><BR/>From: "Belay, Ermias"<BR/>To:<BR/>Cc: "Race, Richard (NIH)" ; ; "Belay,<BR/>Ermias"<BR/>Sent: Monday, September 30, 2002 9:22 AM<BR/>Subject: RE: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG HUNTERS<BR/><BR/>Dear Sir/Madam,<BR/>In the Archives of Neurology you quoted (the abstract of which was<BR/>attached to your email), we did not say CWD in humans will present like<BR/>variant CJD.<BR/><BR/>That assumption would be wrong. I encourage you to read the whole<BR/>article and call me if you have questions or need more clarification<BR/>(phone: 404-639-3091). Also, we do not claim that "no-one has ever been<BR/>infected with prion disease from eating venison." Our conclusion stating<BR/>that we found no strong evidence of CWD transmission to humans in the<BR/>article you quoted or in any other forum is limited to the patients we<BR/>investigated.<BR/><BR/>Ermias Belay, M.D.<BR/>Centers for Disease Control and Prevention<BR/><BR/><BR/>full text ;<BR/><BR/>http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=1165<BR/><BR/><BR/>SEE STEADY INCREASE IN SPORADIC CJD IN THE USA FROM<BR/>1997 TO 2006. SPORADIC CJD CASES TRIPLED, with phenotype<BR/>of 'UNKNOWN' strain growing. ...<BR/><BR/>http://www.cjdsurveillance.com/resources-casereport.html<BR/><BR/><BR/>Diagnosis and Reporting of Creutzfeldt-Jakob Disease<BR/><BR/>Singeltary, Sr et al. JAMA.2001; 285: 733-734.<BR/><BR/>http://jama.ama-assn.org/http://www.neurology.org/cgi/eletters/60/2/176#535<BR/><BR/><BR/>BRITISH MEDICAL JOURNAL<BR/><BR/>BMJ<BR/><BR/>http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406<BR/><BR/><BR/>BMJ<BR/><BR/>http://www.bmj.com/cgi/eletters/320/7226/8/b#6117<BR/><BR/><BR/>JOURNAL OF NEUROLOGY<BR/><BR/>MARCH 26, 2003<BR/><BR/>RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob<BR/><BR/>disease in the United States<BR/><BR/>Email Terry S. Singeltary:<BR/><BR/>flounder@wt.net<BR/><BR/>I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to<BR/><BR/>comment on the CDC's attempts to monitor the occurrence of emerging<BR/><BR/>forms of CJD. Asante, Collinge et al [1] have reported that BSE<BR/><BR/>transmission to the 129-methionine genotype can lead to an alternate<BR/><BR/>phenotype that is indistinguishable from type 2 PrPSc, the commonest<BR/><BR/>sporadic CJD. However, CJD and all human TSEs are not reportable<BR/><BR/>nationally. CJD and all human TSEs must be made reportable in every<BR/><BR/>state and internationally. I hope that the CDC does not continue to<BR/><BR/>expect us to still believe that the 85%+ of all CJD cases which are<BR/><BR/>sporadic are all spontaneous, without route/source. We have many TSEs in<BR/><BR/>the USA in both animal and man. CWD in deer/elk is spreading rapidly and<BR/><BR/>CWD does transmit to mink, ferret, cattle, and squirrel monkey by<BR/><BR/>intracerebral inoculation. With the known incubation periods in other<BR/><BR/>TSEs, oral transmission studies of CWD may take much longer. Every<BR/><BR/>victim/family of CJD/TSEs should be asked about route and source of this<BR/><BR/>agent. To prolong this will only spread the agent and needlessly expose<BR/><BR/>others. In light of the findings of Asante and Collinge et al, there<BR/><BR/>should be drastic measures to safeguard the medical and surgical arena<BR/><BR/>from sporadic CJDs and all human TSEs. I only ponder how many sporadic<BR/><BR/>CJDs in the USA are type 2 PrPSc?<BR/><BR/>http://www.neurology.org/cgi/eletters/60/2/176#535<BR/><BR/><BR/>doi:10.1016/S1473-3099(03)00715-1<BR/>Copyright © 2003 Published by Elsevier Ltd.<BR/>Newsdesk<BR/><BR/>Tracking spongiform encephalopathies in North America<BR/><BR/>Xavier Bosch<BR/><BR/>Available online 29 July 2003.<BR/><BR/><BR/>Volume 3, Issue 8, August 2003, Page 463<BR/><BR/><BR/>“My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my<BR/>mom to hvCJD (Heidenhain variant CJD)<BR/>and have been searching for answers ever since. What I have found is that we<BR/>have not been told the truth. CWD<BR/>in deer and elk is a small portion of a much bigger problem.”<BR/>............................<BR/><BR/><BR/>http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/fulltext<BR/><BR/>http://download.thelancet.com/pdfs/journals/1473-3099/PIIS1473309903007151.pdf<BR/><BR/><BR/><BR/>thank you,<BR/><BR/>kindest regards,<BR/><BR/><BR/>Terry S. Singeltary Sr.<BR/>P.O. Box 42<BR/>Bacliff, Texas USA 77518Terry S. Singeltary Sr.https://www.blogger.com/profile/06986622967539963260noreply@blogger.com